Acute and long-term effects of resistance exercise combined with vascular occlusion on muscular function were investigated. Changes in integrated electromyogram with respect to time (iEMG), vascular resistive index, and plasma lactate concentration were measured in five men either during or after elbow flexion exercises with the proximal end of the arm occluded at 0-100 mmHg. The mean iEMG, postexercise hyperemia, and plasma lactate concentration were all elevated with the increase in occlusion pressure at a low-intensity exercise, whereas they were unchanged with the increase in occlusion pressure at high-intensity exercise. To investigate the long-term effects of low-intensity exercise with occlusion, older women (n = 24) were subjected to a 16-wk exercise training for elbow flexor muscles, in which low-intensity [~50-30% one repetition maximum (1 RM)] exercise with occlusion at ~110 mmHg (LIO), low-intensity exercise without occlusion (LI), and high- to medium-intensity (~80-50% 1 RM) exercise without occlusion (HI) were performed. Percent increases in both cross-sectional area and isokinetic strength of elbow flexor muscles after LIO were larger than those after LI (P < 0.05) and similar to those after HI. The results suggest that resistance exercise at an intensity even lower than 50% 1 RM is effective in inducing muscular hypertrophy and concomitant increase in strength when combined with vascular occlusion.
Je comprend toujours pas ce qu'est une occlusion vasculaire des muscles ou comment on fait ca... mais apparement avec ca, meme des charges hyper légères deviennent efficaces pour l'hypertrophie...
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On the other hand, we have shown that a low-intensity resistance exercise (20-50%1RM) combined with vascular occlusion (occlusive resistance training) induced marked increases in size and strength in elbow flexor muscles of old women and in knee extensor muscles of athletes, even if the intensity of exercise was much lower than expected to promote muscular hypertrophy (Takarada et al. 2000b; Takarada et al. 2002). The mechanisms underlying such an effect of externally applied occlusive stimulus have been interpreted as follows: 1), additional recruitment of fast-twitch fibres in a hypoxic condition (Takarada et al. 2000a; Takarada et al. 2000b) 2), stimulated secretion of growth hormone (GH) and norepinephrine (Takarada et al. 2000a).