Forums SuperPhysique

Hey Darrell- I thought, "Hey, why not check Pubmed".. wow almost 80 studies on overtraining and the cns, here are few ones that confirm what were talking about.

Read the last one expecially!

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*this one generally confirms it

Exercise and its effects on the central nervous system.

Anish EJ.

Division of General Internal Medicine, University of Pittsburgh School of Medicine, Room 216 SON Building, 5230 Centre Avenue, Pittsburgh, PA 15232, USA. anishea@upmc.edu.

Exercise can have profound effects on numerous biologic systems within the human body, including the central nervous system (CNS). The inherent complexity of the CNS, and the methodologic difficulties in evaluating its in vivo neurochemistry in humans, provide challenges to investigators studying the impact of exercise on the CNS. As a result, our knowledge in this area of exercise science remains relatively limited. However, advances in research technology are allowing investigators to gain valuable insight into the neurobiologic mechanisms that contribute to the bidirectional communication that occurs between the periphery and the CNS during exercise. This article examines how exercise-induced alterations in the CNS contribute to central fatigue and the overtraining syndrome, and how exercise can influence psychologic wellbeing and cognitive function.

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*this one shows depression can result from Serotonin problems!

Abnormal serotonin reuptake in an overtrained, insomnic and depressed team athlete.

Uusitalo AL, Valkonen-Korhonen M, Helenius P, Vanninen E, Bergstrom KA, Kuikka JT.

Kuopio University Hospital, Department of Clinical Physiology and Nuclear Medicine, Kuopio University Hospital and University of Kuopio, Finland. arja.uusitalo-koskinen@kuh.fi

The purpose of this report is to study serotonin reuptake of the brain in a severely overtrained athlete by using single-photon emission computed tomography (SPECT). A 26-year-old team athlete increased his training volume (by 200 %) and intensity markedly in a new high-level team. After two months, he started to feel continuous fatigue. He had tinnitus in his left ear, he felt disturbing palpitation and had pollacisuria. After four months, he started to suffer from insomnia. He still continued to play for another three months, after which he was unable to play. He could only sleep for 3 to 4 hours per night. Only minor abnormalities could be found in extensive physical and laboratory examinations. The athlete had a severe overtraining state. In the brain SPECT scans, using the specific radioligand for serotonin transporter imaging ( (123)I labelled 2beta-carbomethoxy-3beta-[4-iodophenyl]-nortropane), low activity areas were detected in the midbrain, anterior gingulus, and left frontal and temporo-occipital lobes. In a psychiatric examination, the patient was found to have signs of major depression, which he hardly recognized himself. We conclude, that that the severe overtraining state could have been related to decreased serotonin reuptake in the brain and signs of major depression.

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*this one shows endocrine problems

Heavy load exercise induced dysfunction of immunity and neuroendocrine responses in rats.

Peijie C, Hongwu L, Fengpeng X, Jie R, Jie Z.

Department of Sports Medicine, Shanghai Institute of Physical Education, Shanghai 200438, China. chenpeijie@online.sh.cn

To determine whether immunity and neuroendocrine system is altered by different loads of exercise training in rats, eight-week-old male Sprague-Dawley rats were randomly assigned to one of the three groups: 1) cage control group (CCG); 2) moderate load training (MLT) (swimming at the intensity of 1.4 m/sec water flowing for 60 min per day); 3) heavy load training (HLT) (swimming at the intensity of 1.8 m/sec water flowing for 120 min per day). MLT and HLT rats were assigned to swim for 6 days per week for total of 6 weeks. All rats were sacrificed 36 h after their last training session. Splenocytes were pooled for assay of cell proliferation and neuropeptide contents in the hypothalamus, hypophysis and plasma were determined by radioimmunoassay while glucocorticoid specific binding in intact thymus was measured by radioligand binding assay. All rats were weighed weekly. The results showed that after 6-week training, rat splenocyte proliferation in response to Con A and LPS decreased in HLT rats compared with MLT and CCG rats. In addition, the contents of beta-endorphin, dynorphin A, arginine vasopressin and oxytocin in the hypothalamus, hypophysis and plasma were altered by HLT, as shown by increased plasma concentration of glucocorticoids and decreased glucocorticoids specific binding in intact thymus compared with MLT and CCG. Furthermore, a decreased body mass in HLT rats has been observed. The body mass of HLT rats was significantly lower than that in CCG and MLT rats at the end of the swimming training period. These data suggest that 6-week heavy load training induces the dysfunction of immunity and neuroendocrine responses, which might be one of the underlying mechanisms of immune dysfunction in overtraining.

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>>>+++ THIS ONE FITS THIS THREAD, SHOWS CNS FATIGUE FROM PULLUPS TO FAILURE!!!+++

Motor cortex fatigue in sports measured by transcranial magnetic double stimulation.

Tergau F, Geese R, Bauer A, Baur S, Paulus W, Reimers CD.

Department of Clinical Neurophysiology, University of Gottingen, Germany. f.tergau@med.uni-goettingen.de

PURPOSE: Besides peripheral mechanisms, central fatigue is an important factor limiting the performance of exhausting exercise in sport. The mechanisms responsible are still in discussion. Using noninvasive transcranial magnetic stimulation (TMS) in a double-pulse technique, we sought to assess fatigue of the motor cortex after exhaustive anaerobic strain. METHODS: 23 male subjects (22-52 yr) taking part in the study were requested to accomplish as many pull-ups as possible until exhaustion. The amount of physical lifting work was recorded. Before and immediately after the task, intracortical inhibition (ICI) and facilitation (ICF) were measured by a conditioned-test double-pulse TMS method for the right brachioradialis (BR) and abductor pollicis brevis muscle (APB). RESULTS: After exercise, ICF was significantly reduced in the BR but not in the APB. ICI was not altered. Changes tended to normalize within 8 min after the task. The amount of lifting work accomplished showed significant correlation to the values of ICF reduction (r = 0.73). Moreover, the baseline values of ICF before exercise were also significantly correlated to the lifting work (r = 0.63). CONCLUSIONS: Because double-pulse TMS gives access to the motor cortex independently of spinal or peripheral mechanisms, reduced ICF reflects decreased excitability of interneuronal circuits within the motor cortex. We suggest that ICF measures motor cortex fatigue after exhausting strain specifically for the muscles performing the task. Gamma-aminobutyric acid (GABA)-ergic neurotransmission is possibly involved in the mechanisms mediating central fatigue. Double-pulse TMS may be a useful tool in the control of training in sports as well as in the detection of pathological central fatigue in overreaching and in the prevention of overtraining.

Vincent a écrit:Bcp ont reporté que meme avec 1-2 séries à l'échec par semaine, ils finissent OVERTRAINED en quelques semaines, au début ca va puis... BOOM d'un coup ca casse et il faut des semaines pour s'en remettre. C'est ce qui m'est arrivé avec mes Singles à 1RM j'ai tenu 3 semaines avant de prendre une énorme claque à la 4ème semaine, et de voir toutes mes perfs chuter violemment.

Jusqu'ici le futur attendu de ton passé ne s'est jamais réalisé dans le présent

The key when designing a therapeutic drug is to optimize the concentration so that minimum dosing is required to produce a maximum response. Likewise the key in bodybuilding should be to optimize intensity so that minimum exercise duration and frequency is required to produce a maximum growth response.

is to optimize the concentration so that minimum dosing

Another critical observation is that the therapeutic effect always plateaus so that pushing the dose higher ONLY produces toxicity and never produces any additional therapeutic effect. Thus, there is NO benefit to risking toxicity. Once you cross the threshold from a therapeutic level of stimulus into the toxic range the organism does not produce any more of the desired response and most times the desired response is PREVENTED by the toxic effect. For instance, the pain relieving effects of Ibuprofen max out at about 400mg, as dosages go higher there is no more pain relief but you may burn a hole in your stomach lining.

Je la trouve très bien ton explication DX !

donc ma question est comment faite vous en pratique pour savoir si vous etes ou non en surentrainement ou sur la voie quels signes avant coureurs utilisez vous

Eponge a écrit:Pour moi le surentrainement est principalement nerveux. Un signe qui ne trompe pas c'est la FC au réveil plus haute.

si tu progresses en étant surentrainé c'est que tu ne l'es pas, du moins les systèmes utilisés dans le strength training ne le sont pas.

Guenael E. a écrit:En fait j'ai l'impression que plus encore que de la force c'est de l'explosivité que je perd en période de surentrainement.

Le vrai sur-entrainement